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On the viral status of SARS-CoV-2: Dr. Meryl Nass on Jon Rappoport’s thesis

From Meryl Nass:

Dr. Thomas Cowan believes that no viruses exist.  Dr. Andrew Kaufman is science-light. Those are Mr. Rappoport’s guiding lights.

Why did that CDC document written in February say CDC had no quantifiable virus samples available?  Because CDC was scamming the US citizenry by creating a faulty test for Covid (and they were quickly informed that it was faulty, but they did not fix it) and refusing to allow anyone else to offer a valid test, until Feb 29.  Of course they had the virus.  But they were not sharing their information nor any samples with anyone until forced, on Feb 29.  I suspect this was part of a plan to allow the virus to spread through the US in the absence of a means to detect it with tests.  But perhaps it was simply gross incompetence and hubris.

You can call SARS-2 anything you want, but it acts like a virus.  But it is transmitted between people and some animals, whether you choose to believe it is alive or dead.  It is grown in labs. It is transmitted between animals in labs.   It can be destroyed (I call it killed) in a broth, petri dish, test tube with a number of drugs.  And its relatives were experimented on in labs, after the progenitors were taken from bats.  And you have to grow them (viruses or whatever you call them) in labs in order to experiment on them.

SARS-2 grows in cells in the back of the nose and throat at first, then moves into more cell types and can kill you by initiating cell death, cytokine storm, hyoercoagulability.

No poisons do that.  It can only happen if the virus GROWS and MULTIPLIES.

Here are some references, and I have included the beginning of the 3’d reference so you can see for yourselves if this is a virus, has been isolated, cultured, etc.

Akst J. Australian Lab Cultures New Coronavirus as Infections Climb. The Scientist. https://www.the-scientist.com/news-opinion/australian-lab-cultures-new-coronavirus-as-infections-climb-67031

2.  CDC has grown the COVID-19 virus in cell culture, which is necessary for further studies, including for additional genetic characterization. The cell-grown virus was sent to NIH’s BEI ResourcesRepository for use by the broad scientific community.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7543926/

Introduction
Virus culture has been regarded as the reference standard of diagnostics for decades, as it allows for identification and isolation of active, replicating virus.1 However, more rapid and sensitive molecular techniques, typically nucleic acid amplification tests (NAAT), such as real-time polymerase chain reaction (PCR), are now the major routine diagnostic tests used in virology diagnostic laboratories. Particularly with a novel or emerging virus such as severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), there are certain circumstances where virus isolation for diagnostic and research purposes remains important, including:

1.To test convalescent sera for neutralising potential, for example as therapeutics for coronavirus disease 2019 (COVID-19) patients in intensive care units.

2.To determine whether infectious virus is present, particularly to inform return to work for previously infectious individuals such as health care workers; individuals with persistent PCR positive results on serial follow-up samples for viral clearance purpose; or when to discontinue transmission-based precautions for patients.2

3.As first line testing for SARS-CoV-2 inactivation efficacy of potential preventative or therapeutic compounds.

4.For use as positive controls in the evaluation of molecular assays.3

The first culture of SARS-CoV-2 internationally was reported by Caly and colleagues in Melbourne, Australia on 28 January 2020 and the cultured virus was rapidly shared globally with other researchers.4 The World Health Organization declared the novel coronavirus a virus of Public Health Emergency of International Concern shortly afterwards on 30 January, and a pandemic on 11 March 2020.

Initial studies of SARS-CoV-2 virus culture were performed using the monkey kidney cell lines Vero-CCL81 and Vero E6.4 , 5 However, various cell lines have been reported as able to sustain SARS-CoV-2 growth and offer a closer approximation to the human immune response. We review here the growth of the virus in existing standard (monkey and human) and engineered cell lines. We discuss the utility of human cell lines in virus culture, and the potential for using these in human immunological and other studies.

SARS-CoV-2 virus isolation
The SARS-CoV-2 virus requires the angiotensin converting enzyme 2 (ACE2) receptor6 for entry into the host cell. This receptor is expressed in lung epithelial cells as well as endothelial cells lining the arteries, veins, capillaries, small intestine, testes, renal tissue and cardiovascular tissue.789 Infection of the host cell also relies on priming of the SARS-CoV-2 spike protein by the transmembrane serine protease (TMPRSS).6 The ACE2 receptor is also used as the receptor for both SARS-CoV and the related human respiratory alphacoronavirus NL63.8

Clinical samples being collected for SARS-Cov-2 nucleic acid amplification tests are upper respiratory tract samples, typically sampling both the nose/nasopharyngeal and throat (oropharyngeal) with (preferably) flocked swabs, as recommended by the Australian Public Health Laboratory Network10 and World Health Organization.11 Lower respiratory tract samples including sputum (if produced) and bronchoalveolar lavage are collected if the lower respiratory tract is suspected to be involved, although risk of virus aerosolisation is higher.11 The virus is detectable by reverse transcription quantitative PCR (RT-qPCR) in the stool of ∼30% patients, and while this may not be infectious, SARS-CoV-2 in one stool sample from a Chinese patient who died from COVID-19 was reported to be culturable after second round passage.12 The virus is not commonly detected in urine; only one of nine patients had a very low level of SARS-CoV-2 in urine detected by real-time PCR in a study by Peng and colleagues.13

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